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Suhagra, like many other medications used to treat erectile dysfunction (ED), contains sildenafil citrate as its active ingredient. The primary mechanism of action of Suhagra is the inhibition of an enzyme called phosphodiesterase type 5 (PDE5). Heres how it works: Smooth Muscle Relaxation: During sexual arousal, nitric oxide (NO) is released in the erectile tissue of the penis. Nitric oxide activates an enzyme called guanylate cyclase, which leads to an increase in cyclic guanosine monophosphate (cGMP) levels. cGMP Signaling: Increased levels of cGMP relax the smooth muscle cells lining the blood vessels in the penis, allowing them to dilate. [b][url=https://www.flatmeds.com/product/suhagra-100-mg/]Suhagra[/url][/b] dilation increases blood flow to the erectile tissue, resulting in an erection. Role of PDE5: Phosphodiesterase type 5 (PDE5) is an enzyme that breaks down cGMP, leading to the cessation of the erection. In individuals with erectile dysfunction, PDE5 activity is often increased, leading to insufficient levels of cGMP for adequate erectile function. Inhibition of PDE5: Sildenafil, the active ingredient in Suhagra, selectively inhibits PDE5, preventing it from breaking down cGMP. This inhibition prolongs the effects of nitric oxide and cGMP, allowing for increased blood flow to the penis and improved erectile function. By inhibiting PDE5, Suhagra enhances the natural physiological process of erection, helping men with ED achieve and maintain satisfactory erections during sexual activity. Its important to note that sexual arousal is still required for Suhagra to be effective, as the medication does not induce an erection on its own.
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